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Melanocortin Agonist

Melanotan-II

MT-II · MT-2 · Cyclic α-MSH Analogue

Melanotan-II is a synthetic cyclic heptapeptide analogue of α-melanocyte-stimulating hormone (α-MSH), developed at the University of Arizona in the 1980s as a photoprotection research compound. Unlike the endogenous α-MSH, the cyclic backbone confers metabolic stability and extends the half-life sufficient for once-daily research dosing. MT-II is broadly active across MC1R, MC3R, MC4R and MC5R — the broad receptor profile underlies both its melanogenic (pigmentation) research applications and the body of MC3R/MC4R-mediated effects on appetite, behaviour and sexual function.

Molecular Profile
SequenceAc-Nle-cyclo[Asp-His-D-Phe-Arg-Trp-Lys]-NH₂
ClassNon-selective MCR Agonist
ReceptorsMC1R · MC3R · MC4R · MC5R
Mol. Weight1024.19 Da
CAS Number121062-08-6
Half-life~33 hours (extended)
OriginUniv. of Arizona, 1980s
PigmentationMC1R AgonismPhotoprotectionAppetite ResearchCyclic PeptideMelanocortin Family
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Laboratory Research Compound — For In Vitro Use Only
This compound is supplied by RS Bio Labs solely as a laboratory research material for use by qualified scientific personnel in in vitro research settings. It is NOT approved, intended, or authorised for human consumption, self-administration, diagnostic, therapeutic, or veterinary use of any kind. All research findings referenced on this page are from preclinical models (cell culture, animal studies) unless explicitly stated otherwise. Preclinical data does not establish safety or efficacy in humans. RS Bio Labs makes no medical or health claims.

Mechanism of Action

Melanotan-II activates all four functional melanocortin receptors. MC1R activation drives the eumelanin/pheomelanin switch in melanocytes — the principal pathway behind its long-standing photoprotection research role. MC3R and MC4R activation in the hypothalamus engages the same satiety and sexual-motivation circuitry that distinguishes the related PT-141 compound; MC5R activation has been studied in lacrimal and sebaceous gland models.

The C-terminal amide group (absent in PT-141) and intact cyclic backbone account for MT-II's substantially longer half-life and broader receptor activity compared with endogenous α-MSH, which is rapidly degraded.

Photoprotection research conducted at the University of Arizona established that MT-II-induced eumelanin synthesis provides quantifiable UV-A and UV-B absorption across treated skin in preclinical models — the original research rationale.

01
MC1R Eumelanin Switch
Drives melanocyte-stimulating hormone activity at the cutaneous MC1R, promoting eumelanin synthesis — the pigmentation pathway most studied in skin-research models.
02
MC4R Central Activity
Engages hypothalamic MC4R, with downstream effects on appetite, satiety and reward circuitry comparable to PT-141 but accompanied by additional MC1R and MC5R activity.
03
Long Plasma Half-Life
C-terminal amide and cyclic backbone extend circulation to ~33 hours — substantially longer than endogenous α-MSH or first-generation analogues.

Photoprotection Research

Originally synthesised at the University of Arizona by Dr Victor Hruby and colleagues with the explicit research objective of identifying a photoprotection compound. Subsequent preclinical research has documented dose-dependent skin darkening (eumelanin synthesis) and measurable UV-absorption changes in treated tissue. No regulated medicinal product currently exists in this class.

Distinct from the more selective PT-141 (which omits the C-terminal amide), MT-II's broad receptor profile makes it the standard research probe for non-selective melanocortin biology.

Appetite & Behaviour Pathways

Beyond the dermal research, MT-II's central MC4R activity has been characterised in appetite-suppression and sexual-motivation preclinical models. The same circuitry underpins the PT-141 indication; MT-II is studied where broader receptor coverage is the research aim.

Adverse-event findings consistent across the literature include transient nausea, facial flushing, spontaneous penile erection in male research subjects, and pigment darkening of pre-existing nevi — the latter is the reason photoprotection development stalled.

Key Published Research
Melanocortin Analogues for Tanning and Photoprotection
Pigment Cell & Melanoma Research · Hadley & Dorr · 2006
Foundational review of the Arizona-developed melanocortin analogues including MT-II. Documents dose-dependent eumelanin synthesis, UV-absorption changes and the receptor-pharmacology profile that distinguishes MT-II from later selective analogues.
Melanotan-II: A Cyclic α-MSH Analogue Stimulating Skin Pigmentation in Caucasian Subjects
British Journal of Dermatology · Dorr et al. · 2004
Small clinical study in 13 caucasian research subjects documenting measurable skin-pigmentation responses to MT-II dosing across a 10-day protocol. Established the dose-response relationship that subsequent photoprotection research built on.
Adverse Events Associated with Unregulated Use of Melanotan-II
Journal of the European Academy of Dermatology and Venereology · 2009
Review of reported adverse events from unregulated MT-II use in cosmetic tanning contexts. Pigmented-lesion darkening, transient nausea, facial flushing and case reports of nevus dysplasia documented — collectively the reason MT-II remains an unregulated research compound rather than a clinical product.
Research Context: Melanotan-II is an unregulated research compound. It has not been approved by the MHRA, FDA or any other regulatory authority for any medicinal indication. RS Bio Labs supplies it as a research-grade laboratory compound intended exclusively for in vitro scientific research — not for human consumption, self-administration, veterinary use, or therapeutic application. This profile is provided for educational and scientific reference only.