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Long-Acting Amylin Analogue

Cagrilintide

AM833 · Long-Acting Amylin Receptor Agonist

Cagrilintide is a synthetic long-acting amylin analogue developed by Novo Nordisk, engineered for once-weekly subcutaneous dosing. Amylin is a 37-amino-acid neuroendocrine peptide co-secreted with insulin from pancreatic β-cells; it regulates postprandial glucagon, gastric emptying and satiety. Cagrilintide is studied both as a monotherapy weight-management compound and — more prominently — as the partner of semaglutide in the dual-mechanism CagriSema research programme, which has reported some of the largest weight-loss effects observed in the GLP-1 / amylin combination class.

Molecular Profile
SynonymAM833
ClassLong-Acting Amylin Analogue
Half-life~7 days
ModificationAcylated, plasma-stable
ReceptorsAMY₁ · AMY₂ · AMY₃
DosingWeekly (research)
OriginatorNovo Nordisk
Weight ManagementAmylin PathwaySatiety ResearchCagriSema ProgrammeGastric EmptyingPostprandial Glucagon
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Laboratory Research Compound — For In Vitro Use Only
This compound is supplied by RS Bio Labs solely as a laboratory research material for use by qualified scientific personnel in in vitro research settings. It is NOT approved, intended, or authorised for human consumption, self-administration, diagnostic, therapeutic, or veterinary use of any kind. All research findings referenced on this page are from preclinical models (cell culture, animal studies) unless explicitly stated otherwise. Preclinical data does not establish safety or efficacy in humans. RS Bio Labs makes no medical or health claims.

Mechanism of Action

Cagrilintide is an agonist at the three amylin receptors AMY₁, AMY₂ and AMY₃, which are heterodimeric complexes of the calcitonin receptor with receptor activity-modifying proteins 1, 2 and 3. The principal sites of action relevant to weight-management research are the area postrema and dorsal motor nucleus of the vagus in the brainstem, where amylin signalling drives satiety and slows gastric emptying.

The acylated long-acting backbone (an analogue strategy similar to that used for semaglutide and tirzepatide) extends the plasma half-life to approximately 7 days, supporting once-weekly research dosing. Compared with shorter-acting amylin analogues (pramlintide), cagrilintide's pharmacokinetic profile permits dosing frequencies compatible with GLP-1 partner compounds.

The CagriSema research thesis is that amylin and GLP-1 act on partially non-overlapping satiety pathways — amylin acting predominantly via the brainstem, GLP-1 via additional hypothalamic and vagal circuits — and combining them produces additive or synergistic weight-loss effects exceeding either monotherapy.

01
AMY₁/₂/₃ Receptor Agonism
Activates the three amylin receptor subtypes (calcitonin receptor + RAMP1/2/3 heterodimers) at brainstem nuclei involved in satiety and gastric-emptying control.
02
Brainstem Satiety Signalling
Engages the area postrema and dorsal motor nucleus of the vagus — anatomically distinct from the hypothalamic and arcuate-nucleus circuits engaged by GLP-1 agonists, supporting the combination thesis.
03
Once-Weekly Pharmacokinetics
Acylated backbone extends half-life to ~7 days, compatible with the dosing cadence of GLP-1 partners semaglutide and tirzepatide.

CagriSema Combination Research

The CagriSema programme (cagrilintide 2.4 mg + semaglutide 2.4 mg) is the most studied amylin/GLP-1 combination in development. A Phase 2 trial (Lancet, 2021) in adults with overweight/obesity reported approximately 17% weight loss at 20 weeks with the combination versus approximately 8% with semaglutide alone — establishing the additive-effect thesis.

The pivotal Phase 3 REDEFINE programme continues to read out. The combination is widely viewed as a candidate to challenge tirzepatide on weight-loss magnitude.

Amylin Pathway Research

Cagrilintide monotherapy at 2.4 mg weekly produced approximately 10% weight loss in Phase 2 (Diabetes, Obesity & Metabolism, 2021) — substantial but less than GLP-1 monotherapy. The compound's interest is primarily as a combination partner rather than as a standalone weight-management compound.

Beyond weight management, amylin pathway research has applications in postprandial glucagon control in type 1 diabetes (the original pramlintide indication) and emerging interest in neurodegenerative research where amylin-pathway dysregulation has been documented in Alzheimer's disease preclinical models.

Key Published Research
Effect of Cagrilintide Alone or in Combination with Semaglutide on Body Weight (CagriSema Phase 2)
The Lancet · Enebo et al. · 2021
Phase 2 trial in 92 adults with overweight randomised to cagrilintide alone, semaglutide alone, or the combination over 20 weeks. The combination produced approximately 17% weight loss vs ~8% with semaglutide alone and ~10% with cagrilintide alone — establishing the additive-effect thesis driving the Phase 3 REDEFINE programme.
Cagrilintide, a Long-Acting Amylin Analog: A Randomized Dose-Finding Phase 2 Trial
Diabetes, Obesity & Metabolism · 2021
26-week dose-finding study of cagrilintide monotherapy across 0.16–4.5 mg doses. Established the 2.4 mg dose later used in CagriSema. Mean weight reductions of approximately 10% at the top dose; adverse-event profile dominated by transient gastrointestinal effects consistent with the amylin class.
Amylin and the GLP-1 / Amylin Combination Strategy: Pathway and Pharmacology
Trends in Endocrinology & Metabolism · 2022
Review of the amylin pathway and the pharmacological rationale for combining amylin agonism with GLP-1 agonism. Documents the partially non-overlapping satiety circuitry and explains why combination effects exceed monotherapy in published preclinical and clinical research.
Research Context: Cagrilintide is an investigational compound in active Phase 3 clinical development by Novo Nordisk. It has not been approved by the MHRA, FDA or any other regulatory authority. RS Bio Labs supplies it as a research-grade laboratory compound for in vitro scientific use only. It is not for human consumption, self-administration, veterinary use, or therapeutic application. This profile is for educational and scientific reference only.